Professor Sucharit Bhakdi Unveils a New Theory on Post-mRNA Vaccine Illness
/ 2026 / … In a surprise addition to the programme,
Professor Sucharit Bhakdi, former chair of the Institute of Medical Microbiology at the University of Mainz, chose the UK Column 20th Anniversary On Location audience to present a hypothesis he had not shared publicly before.
Professor Bhakdi begins with his life and career, from his childhood in Bern, Cairo and Bangkok, his medical training in Bonn, to his research at the Max Planck Institute of Immunobiology. He describes how the corona years led him to Gustave Le Bon’s work on crowd psychology and to question decades of his own teaching on vaccination.
He mentions books he has co-authored, including a German-language title with Karina Reiss and Klaus Köhnlein, and a forthcoming English edition of a compendium written with former Pfizer toxicologist Helmut Sterz, to be published by Skyhorse.
According to Bhakdi’s hypothesis, the mRNA is taken up by endothelial cells in the smallest blood vessels. The production of foreign proteins (in this case, the spike protein – but any other would have the same effect) leads to an attack on and damage to the vascular endothelium, thereby causing disruptions to the blood supply and the death of surrounding cells. These include oligodendrocytes, which are essential for maintaining the myelin sheaths around nerves. The breakdown of the myelin sheaths leads to disturbances in nerve conduction and to symptoms characteristic of classic degenerative demyelinating diseases such as Alzheimer’s, multiple sclerosis, Parkinson’s and many others – symptoms which we observe in vaccine-injured individuals.
The breakdown products of the myelin sheaths have a unique property: they activate the complement system, so that the surrounding oligodendrocytes come under attack from complement. Oligodendrocytes have remarkably few protective proteins against these complement attacks, resulting in a selfperpetuating cycle of destruction. According to this hypothesis, this process occurs in all degenerative neurological diseases and explains their relentless progression.
He closes by discussing curcumin as a possible complement inhibitor. He cites early anecdotal reports from a Norwegian doctor and from friends he is supporting.
Professor Bhakdi presents this explicitly as an untested hypothesis and invites scientists to try to disprove it.
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